receptor 4 tlr4 Search Results


tlr4 human recombinant protein  (MedChemExpress)
94
MedChemExpress tlr4 human recombinant protein
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Tlr4 Human Recombinant Protein, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/TLR4%2C+Human/pmc11726454-374-0-5
Average 94 stars, based on 1 article reviews
tlr4 human recombinant protein - by Bioz Stars, 2026-10
94/100 stars
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tlr 4  (Elabscience Biotechnology)
93
Elabscience Biotechnology tlr 4
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Tlr 4, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Rat+TLR4+(Toll-Like+Receptor+4)+ELISA+Kit/pmc10788501-138-50-59
Average 93 stars, based on 1 article reviews
tlr 4 - by Bioz Stars, 2026-10
93/100 stars
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anti tlr4 antibody  (Proteintech)
96
Proteintech anti tlr4 antibody
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Anti Tlr4 Antibody, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/TLR4+Antibody/pmc11315675-24-44-48
Average 96 stars, based on 1 article reviews
anti tlr4 antibody - by Bioz Stars, 2026-10
96/100 stars
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mrna expressions  (MedChemExpress)
97
MedChemExpress mrna expressions
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Mrna Expressions, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Toll-Like+Receptor+4+Antibody/pmc09363708-118-1-17
Average 97 stars, based on 1 article reviews
mrna expressions - by Bioz Stars, 2026-10
97/100 stars
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e el m2417  (Elabscience Biotechnology)
93
Elabscience Biotechnology e el m2417
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
E El M2417, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Mouse+TLR4+(Toll-Like+Receptor+4)+ELISA+Kit/pmc12414226-77-11-12
Average 93 stars, based on 1 article reviews
e el m2417 - by Bioz Stars, 2026-10
93/100 stars
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tlr4  (ProSci Incorporated)
90
ProSci Incorporated tlr4
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Tlr4, supplied by ProSci Incorporated, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/TLR4+Antibody/pmc03069425-62-5-11
Average 90 stars, based on 1 article reviews
tlr4 - by Bioz Stars, 2026-10
90/100 stars
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elisa kit  (Elabscience Biotechnology)
94
Elabscience Biotechnology elisa kit
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Elisa Kit, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Human+TLR4+(Toll-Like+Receptor+4)+ELISA+Kit/pm41598496-81-13-23
Average 94 stars, based on 1 article reviews
elisa kit - by Bioz Stars, 2026-10
94/100 stars
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yeast  (Cusabio)
93
Cusabio yeast
a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and <t>TLR4</t> was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.
Yeast, supplied by Cusabio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Recombinant+Mouse+Toll-like+receptor+4(Tlr4)%2Cpartial/pm35963236-291-54-55
Average 93 stars, based on 1 article reviews
yeast - by Bioz Stars, 2026-10
93/100 stars
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rat tlr4 elisa kit  (Cusabio)
93
Cusabio rat tlr4 elisa kit
Tongue tissue <t>TLR4</t> and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)
Rat Tlr4 Elisa Kit, supplied by Cusabio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Rat+toll-like+receptor+4+(TLR4)+ELISA+Kit/pmc12659584-125-10-14
Average 93 stars, based on 1 article reviews
rat tlr4 elisa kit - by Bioz Stars, 2026-10
93/100 stars
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receptor 4  (Biorbyt)
93
Biorbyt receptor 4
Tongue tissue <t>TLR4</t> and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)
Receptor 4, supplied by Biorbyt, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/TLR4+(Toll+Like+Receptor+4)+antibody/pm35775747-92-27-47
Average 93 stars, based on 1 article reviews
receptor 4 - by Bioz Stars, 2026-10
93/100 stars
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tlr4  (Boster Bio)
93
Boster Bio tlr4
Tongue tissue <t>TLR4</t> and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)
Tlr4, supplied by Boster Bio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/receptor+4+tlr4/Anti-TLR4+(S441)+Antibody/pm41776247-283-34-44
Average 93 stars, based on 1 article reviews
tlr4 - by Bioz Stars, 2026-10
93/100 stars
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human toll like receptor 4 elisa kit  (Cusabio)
93
Cusabio human toll like receptor 4 elisa kit
Tongue tissue <t>TLR4</t> and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)
Human Toll Like Receptor 4 Elisa Kit, supplied by Cusabio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and TLR4 was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Journal: Nature Microbiology

Article Title: Gut–liver translocation of pathogen Klebsiella pneumoniae promotes hepatocellular carcinoma in mice

doi: 10.1038/s41564-024-01890-9

Figure Lengend Snippet: a , Representative SEM (top) and TEM (bottom) images of Hep3B and Huh7 HCC cell lines after co-culture with K. pneumoniae (MOI = 10). After co-culture of HCC cells with K. pneumoniae , cell lysate was spread on BHI agar for bacterial culture. n = 3 independent experiments with similar results. b , Effect of pasteurized K. pneumoniae (MOI = 10) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). c , Effect of pasteurized K. pneumoniae (MOI = 10) on PCNA expression in HCC cells. n = 3 (Ctrl), 6 (pasteurized KP). d , Screening of K. pneumoniae adhesins by biotin pull-down assay. e , Hep3B membrane protein was incubated with GST or GST–PBP1B together with GST magnetic beads for GST pull-down assay. Corresponding bands in GST–PBP1B and Hep3B cell membrane protein groups were subjected to mass spectrometry analysis. In d and e , n = 3 independent experiments with similar results. f , Binding affinity between PBP1B and TLR4 was detected using SPR. K d , dissociation constant. g , Representative structure of PBP1B and TLR4 after molecular docking (left) and the residues involved in the interaction between PBP1B with TLR4 (right). The structure of TLR4 is coloured pink while the PBP1B is coloured yellow. The light green dash represents hydrogen bond. h , TLR4 from Hep3B and Huh7 cell lysates was pulled down by GST–PBP1B according to the GST pull-down assay. i , Immunoprecipitation of TLR4 from Hep3B or Huh7 cells lysates validated the binding between TLR4 and GST–PBP1B. In h and i , n = 3 independent experiments with similar results. j , Effect of PBP1B (0.05 μM) on HCC cell proliferation (top) ( n = 3 biologically independent samples), colony formation (middle) ( n = 3 biologically independent samples) and growth of HCC patient-derived organoid (bottom) ( n = 20 biologically independent samples). n = 3 independent experiments with similar results. k , Effect of PBP1B (0.05 μM) on TLR4, MyD88, p-P65, P65 and PCNA protein expression in HCC cells, as determined by western blot. n = 3 biologically independent samples. l , m , TLR4i (30 μM) abolished the effect of PBP1B on HCC cell proliferation ( l ) and colony formation ( m ). n = 3 biologically independent samples. n , Effect of K. pneumoniae on TLR4 expression in liver tissues of both germ-free and SPF mice with or without DEN treatment by IHC staining. In b , c , j – m , data are presented as mean ± s.e.m. Each data point in bar plots represents one mouse. Cell proliferation was analysed using two-way ANOVA. In b , c , j , k , m , comparisons between two groups were analysed using Student’s t -test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Article Snippet: TLR4 human recombinant protein (HY-P73586, MCE) was immobilized on a Series S Sensor Chip CM5 (29149603, GE Healthcare) using an amine coupling kit (BR-1000-50, GE Healthcare) with a flow rate of 10 μl min −1 , resulting in a final ligand coupling of ~180 response units.

Techniques: Co-Culture Assay, Derivative Assay, Expressing, Pull Down Assay, Membrane, Incubation, Magnetic Beads, Mass Spectrometry, Binding Assay, Immunoprecipitation, Western Blot, Immunohistochemistry

(a) Design of K. oxytoca on K. pneumoniae induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=6; GFD/EC group n=6; GFD/KO group n=6; GFD/KP group n=7; GFD/KOP group n=8) and relevant results (b–e) . (b) Images of liver gross morphology (yellow circles indicate tumor area), and H&E with tumor incidence, tumor number and tumor burden. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (c) Alcian blue staining, E-Cad, CLDN 3, CLDN 1 IHC, and Cy3-conjugated EUB338 probe detection with quantitative analysis. n=6 biologically independent samples. (d) Gut permeability assay using FITC-dextran. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (e) Live bacteria culture of liver tissues on blood agar plates with quantitative analysis. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (f) Design of TLR4i on K. pneumoniae induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=6; GFD/EC group n=6; GFD/KP group n=7; GFD/KPi group n=7) and relevant results (g-k) . (g) Images of liver gross morphology (yellow circles indicate tumor area), and H&E with tumor incidence, tumor number and tumor burden. n=6 (GFD/PBS), 6 (GFD/EC), 7 (GFD/KP), 7 (GFD/KPi). (h) E-Cad IHC and Cy3-conjugated EUB338 probe (GFD/PBS group), E. coli probe (GFD/EC), K. pneumoniae probe (GFD/KP) and GFD/KPi groups) detection of colon tissues with quantitative analysis. n=6 biologically independent samples. (i) Gut permeability assay using FITC-dextran. n=6 biologically independent samples. (j) Live bacteria culture of liver tissues on blood agar plates with quantitative analysis. n=6 (GFD/PBS), 6 (GFD/EC), 7 (GFD/KP), 7 (GFD/KPi). (k) Cy3-conjugated conjugated EUB338 probe (GFD/PBS group), E. coli probe (GFD/EC), K. pneumoniae probe (GFD/KP) and GFD/KPi groups) FISH, TLR4, PCNA, Ki-67 IHC, and Sirius red staining of liver sections. n=6 biologically independent samples. Data (excluding tumor incidence and liver with live bacteria) are presented as mean ± SEM. Each data point in bar plots represents one mouse. Tumor incidence and Liver with live bacteria were calculated using Fisher’s exact test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Journal: Nature Microbiology

Article Title: Gut–liver translocation of pathogen Klebsiella pneumoniae promotes hepatocellular carcinoma in mice

doi: 10.1038/s41564-024-01890-9

Figure Lengend Snippet: (a) Design of K. oxytoca on K. pneumoniae induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=6; GFD/EC group n=6; GFD/KO group n=6; GFD/KP group n=7; GFD/KOP group n=8) and relevant results (b–e) . (b) Images of liver gross morphology (yellow circles indicate tumor area), and H&E with tumor incidence, tumor number and tumor burden. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (c) Alcian blue staining, E-Cad, CLDN 3, CLDN 1 IHC, and Cy3-conjugated EUB338 probe detection with quantitative analysis. n=6 biologically independent samples. (d) Gut permeability assay using FITC-dextran. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (e) Live bacteria culture of liver tissues on blood agar plates with quantitative analysis. n=6 (GFD/PBS), 6 (GFD/EC), 6 (GFD/KO) 7 (GFD/KP), 8 (GFD/KOP). (f) Design of TLR4i on K. pneumoniae induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=6; GFD/EC group n=6; GFD/KP group n=7; GFD/KPi group n=7) and relevant results (g-k) . (g) Images of liver gross morphology (yellow circles indicate tumor area), and H&E with tumor incidence, tumor number and tumor burden. n=6 (GFD/PBS), 6 (GFD/EC), 7 (GFD/KP), 7 (GFD/KPi). (h) E-Cad IHC and Cy3-conjugated EUB338 probe (GFD/PBS group), E. coli probe (GFD/EC), K. pneumoniae probe (GFD/KP) and GFD/KPi groups) detection of colon tissues with quantitative analysis. n=6 biologically independent samples. (i) Gut permeability assay using FITC-dextran. n=6 biologically independent samples. (j) Live bacteria culture of liver tissues on blood agar plates with quantitative analysis. n=6 (GFD/PBS), 6 (GFD/EC), 7 (GFD/KP), 7 (GFD/KPi). (k) Cy3-conjugated conjugated EUB338 probe (GFD/PBS group), E. coli probe (GFD/EC), K. pneumoniae probe (GFD/KP) and GFD/KPi groups) FISH, TLR4, PCNA, Ki-67 IHC, and Sirius red staining of liver sections. n=6 biologically independent samples. Data (excluding tumor incidence and liver with live bacteria) are presented as mean ± SEM. Each data point in bar plots represents one mouse. Tumor incidence and Liver with live bacteria were calculated using Fisher’s exact test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Article Snippet: TLR4 human recombinant protein (HY-P73586, MCE) was immobilized on a Series S Sensor Chip CM5 (29149603, GE Healthcare) using an amine coupling kit (BR-1000-50, GE Healthcare) with a flow rate of 10 μl min −1 , resulting in a final ligand coupling of ~180 response units.

Techniques: Staining, Permeability, Bacteria

(a) Design of TLR4i on HCC-FMT induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=8; GFD/HCC-FMT group n=15; GFD/HCC-FMTi group n=10) and relevant results (b–g) . TLR4i was daily gavaged in this experiment. (b) Representative images of liver gross morphology (yellow circles indicate tumor area), and H&E staining of mice liver sections with tumor incidence, tumor number, and tumor burden. n=8 (GFD/PBS), 15 (GFD/HCC-FMT), 10 (GFD/HCC-FMTi). (c) Representative bacterial culture of liver tissues under anaerobic and aerobic conditions with quantitative analysis. n=8 (GFD/PBS), 15 (GFD/HCC-FMT), 10 (GFD/HCC-FMTi). From left to right and top to bottom, the culture plates are blood agar plate, chocolate blood agar plate, MacConkey agar plate, and Columbia blood agar plate, respectively. Gut permeability assay using 500 kDa FITC-dextran. n=6 biologically independent samples. (d) Representative pictures of Alcian blue staining, E-Cad IHC staining, and Cy3-conjugated EUB338 probe FISH detection of colon tissues with quantitative analysis. n=6 biologically independent samples. (e) Cy3-conjugated EUB338 probe FISH, Cy3-conjugated K. pneumoniae probe FISH, TLR4, PCNA, Ki-67 IHC of liver sections. n=6 biologically independent samples. (f) α-SMA, Desmin IHC staining, and Sirius red staining of liver sections. n=6 biologically independent samples. (g) Mouse Cancer Pathway Finder Array and Inflammatory Response and Autoimmunity PCR Array of liver tissues. n=3 independent experiments with similar results. Data (excluding tumor incidence, liver with live bacteria and PCR array results) are presented as mean ± SEM. Each data point in bar plots represents one mouse. Tumor incidence and Liver with live bacteria were calculated using Fisher’s exact test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Journal: Nature Microbiology

Article Title: Gut–liver translocation of pathogen Klebsiella pneumoniae promotes hepatocellular carcinoma in mice

doi: 10.1038/s41564-024-01890-9

Figure Lengend Snippet: (a) Design of TLR4i on HCC-FMT induced HCC promotion in germ-free mice with DEN treatment (GFD/PBS group n=8; GFD/HCC-FMT group n=15; GFD/HCC-FMTi group n=10) and relevant results (b–g) . TLR4i was daily gavaged in this experiment. (b) Representative images of liver gross morphology (yellow circles indicate tumor area), and H&E staining of mice liver sections with tumor incidence, tumor number, and tumor burden. n=8 (GFD/PBS), 15 (GFD/HCC-FMT), 10 (GFD/HCC-FMTi). (c) Representative bacterial culture of liver tissues under anaerobic and aerobic conditions with quantitative analysis. n=8 (GFD/PBS), 15 (GFD/HCC-FMT), 10 (GFD/HCC-FMTi). From left to right and top to bottom, the culture plates are blood agar plate, chocolate blood agar plate, MacConkey agar plate, and Columbia blood agar plate, respectively. Gut permeability assay using 500 kDa FITC-dextran. n=6 biologically independent samples. (d) Representative pictures of Alcian blue staining, E-Cad IHC staining, and Cy3-conjugated EUB338 probe FISH detection of colon tissues with quantitative analysis. n=6 biologically independent samples. (e) Cy3-conjugated EUB338 probe FISH, Cy3-conjugated K. pneumoniae probe FISH, TLR4, PCNA, Ki-67 IHC of liver sections. n=6 biologically independent samples. (f) α-SMA, Desmin IHC staining, and Sirius red staining of liver sections. n=6 biologically independent samples. (g) Mouse Cancer Pathway Finder Array and Inflammatory Response and Autoimmunity PCR Array of liver tissues. n=3 independent experiments with similar results. Data (excluding tumor incidence, liver with live bacteria and PCR array results) are presented as mean ± SEM. Each data point in bar plots represents one mouse. Tumor incidence and Liver with live bacteria were calculated using Fisher’s exact test. Unless otherwise stated, statistical significance was calculated using one-way ANOVA. Adjustments were made for multiple comparisons.

Article Snippet: TLR4 human recombinant protein (HY-P73586, MCE) was immobilized on a Series S Sensor Chip CM5 (29149603, GE Healthcare) using an amine coupling kit (BR-1000-50, GE Healthcare) with a flow rate of 10 μl min −1 , resulting in a final ligand coupling of ~180 response units.

Techniques: Staining, Chocolate, Permeability, Immunohistochemistry, Bacteria

Tongue tissue TLR4 and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)

Journal: BMC Oral Health

Article Title: Edaravone attenuates doxorubicin-induced oral mucosal injury via modulation of oxidative stress, inflammatory signaling, and the SIRT1/TLR4/NF-kB/ACE2 axis in rats

doi: 10.1186/s12903-025-07148-y

Figure Lengend Snippet: Tongue tissue TLR4 and proinflammatory cytokines. A TLR4 (ng/mg protein), ( B ) TNF-α (ng/L/g), ( C ) IL-6 (ng/L/g) (mean ± SEM). DOX increased TLR4/TNF-α/IL-6; EDO reduced these levels. Statistics: TLR4/IL-6—ANOVA ± Tukey; TNF-α—Kruskal–Wallis ± Mann–Whitney. Significance: * p < 0.05, ** p < 0.01, *** p < 0.001; ns, not significant (adjusted p ≥ 0.05)

Article Snippet: TLR4 protein levels in tongue tissue were quantified using a rat TLR4 ELISA kit (Cusabio, Cat. No: CSB-E15822r).

Techniques: MANN-WHITNEY